EMF Blocking in addition to EMF Shielding

No matter if you live in a house or apartment, or you just would like to ensure your home is free of electromagnetic fields, there are a number of methods to reduce exposure. One of the easiest is to reduce your use of electronic devices. It is also possible to use EMF block paint to prevent EMF radiation from entering your home. Another way to shield your house against EMF radiation would be to install a shielding canopy for RF. It is a type made of net that has EMF shielding and is used to prevent EMFs from entering a space. Another option is to get your house equipped with an electrical enclosure. These enclosures are known as Faraday cages.

emf guard  have demonstrated that the non-ionizing RF EMF can cause antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to involve down-regulation the cancer stem cell. This may account for the long-term effects observed in certain patients suffering from advanced HCC. However, the mechanism behind AM RF EMF's effect in patients suffering from cancer isn't yet clear.

Effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were examined in mice. The tumors were split into 3 groups. One group did not have exposure to RF EMF. Another group of participants was subjected RF EMF at the same frequency to the one used by humans. In the third, they were subjected to the RF EMF in HCC-specific frequencies. The effect of HCCMF on tumors was assessed against the effect of RCF. The results revealed that cancers treated with HCCMF were significantly shrinking. However, the tumors treated with RCF showed no evidence of shrinkage of the tumor.

The mechanism behind tumour-specific AM RF EMF might be due to the fact that cancer cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects on HCC cells is caused through CACNA1H the protein which mediates tumour-specific Ca2+ influx.  emf block  suggest that CACNA1H could have wider implications in the diagnosis and treatment of various cancers.

The tumours of the control group were not exposed to EMF from RF, and fed a normal mouse diet. The tumours in the HCCMF group were infected with Huh7 cells after they were 5 to 7 weeks old. The tumours were then euthanized when they showed excessive burden.

The tumors of the three groups showed different growth curves. The HCCMF-treated tumors showed a significant decrease in the size of the tumour after eight weeks. However, the tumours that were treated using RCF didn't show reduction in size. The difference was highly significant.  emf guard  treated by RCF were able to show necrosis, which is typical when tumors are that are exposed to RCF. There is a possibility that the necrosis was caused by the lack of oxygen in the larger tumours.

In summary, the results show the fact that AM EMF has anticancer effects in vitro and in the vivo. Several studies have shown the fact that AM RF EMF produces measurable reduction in tumours within HCC patients. There is a possibility that AM RF EMF triggers these effects through CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may cause a lasting influence on the growth of HCC tumours in the vivo.